Pregnancy-induced adaptations in the metabolism of glucose, lipids, and, to a lesser extent, amino acids have been well described in ruminants and other domestic animals. These adaptations are mediated by altered responses in multiple tissues to homeostatic effectors such as insulin and catecholamines in a way that is coordinated to promote the availability of vital nutrients to the developing fetus(es). These phenomena are the hallmarks of homeorhetic regulation of nutrient partitioning in support of a chronic physiological imperative. However, the identity of the factor(s) responsible for homeorhetic modulation of metabolic adaptations to pregnancy remains to be elucidated.

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