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Dorn Spinal Therapy

Spine Healing Therapy

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Clinical Features

The victim of an acute injury of the cervical or upper thoracic area of the spine may go into a state of sympathetic spinal shock (Guttmann 1976; Stauffer 1987). The cord distal to the level of injury enters a state of areflexia. For a short period of time (6-24 h), the victim will exhibit areflexia with hypotonia and a "sympathectomy" effect. The vascular tree dilates and blood accumulates in the peripheral vascula-ture. Typically, the patient's blood pressure drops to 90 mmHg systolic over 50 or 60 mmHg diastolic in the absence of a rapid pulse. Reflexes begin to recover after 24 h and between 48 h and 72 h after the blunt impact the vasculature begins to contract. A patient in spinal shock can die suddenly, especially if high cervical SCI coexists with pontomedullary

Fig. 10.4a-f. Morphology of different biomechanical mechanisms leading to closed spinal cord injuries. a Flexion injury as demonstrated by dorsal hemorrhages in the intervertebral discs between C3/C4 and C4/C5 (arrows); b, c hyperextension caused ventral hemorrhage in the intervertebral disc between C3/C4 and C4/C5 (arrows) and a rupture of the disc C5/C6 (circle) as well as an epidural hemorrhage; d deflexion injury associated with fractures of C4 and C5, a compression of the spinal cord and epidural hemorrhages; e compression caused fractures of several vertebral bodies (arrows): C6, Th1-Th3; f rotation induced asymmetrical fractures of vertebral bodies and their processes associated with compression of the spinal cord

Fig. 10.4a-f. Morphology of different biomechanical mechanisms leading to closed spinal cord injuries. a Flexion injury as demonstrated by dorsal hemorrhages in the intervertebral discs between C3/C4 and C4/C5 (arrows); b, c hyperextension caused ventral hemorrhage in the intervertebral disc between C3/C4 and C4/C5 (arrows) and a rupture of the disc C5/C6 (circle) as well as an epidural hemorrhage; d deflexion injury associated with fractures of C4 and C5, a compression of the spinal cord and epidural hemorrhages; e compression caused fractures of several vertebral bodies (arrows): C6, Th1-Th3; f rotation induced asymmetrical fractures of vertebral bodies and their processes associated with compression of the spinal cord or other brain stem injury (Braakman and Penning 1976; Leestma et al. 1983).

The chronic (and acute) neurological (sensory and motor) deficits depend on the segment of the cord that is injured. In adults, the spinal cord ends at the inferior aspect of the L-1 vertebra. The injured spinal cord segments, therefore, do not correspond with the vertebral bony segment level.

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