Conclusions

Clearly, the variety of adhesion molecules described to date lends itself well to diverse mechanisms of immune regulation. New adhesion molecules are likely to be discovered, and the mechanisms of adhesion molecule function will continue to be further delineated. Given the breadth of this diversity, it will be crucial to begin a closer scrutiny of the structural and signaling requirements of these molecules. A more precise knowledge of these roles may lead to valuable tools that target adhesion molecules for therapeutic intervention in diseases whose etiology includes aberrations of the immune response.

See also: Carbohydrate antigens; CD2; CD44; Chemokines; Chemotaxis; Chemotaxis of lymphocytes; Chemotaxis of macrophages and monocytes; Chemotaxis of neutrophils; Diapedesis; Endothelium; Extracellular matrix; Fibronectin; High endothelial venules; Immune adherence; Immunodeficiency, animal models; Immunoglobulin gene superfamily; Integrins; Intercellular adhesion molecules: ICAM-1, ICAM-2 and ICAM-3; Knockout, genetic; Lectins; Leukocyte adhesion deficiency; Lewis7sialyl-Lewisx (CD15/CD15s); Lymphocyte function-associated antigen 1 (LFA-1); Lymphocyte function-associated antigen 3 (LFA-3); Lymphocyte trafficking; Proliferation, lymphocyte; Second signals for lymphocyte activation; Selectins (CD62-E/LVP); T lymphocyte activation; Yersinia, infection and immunity.

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