Cok

1 Antibody |

Years

Figure 3 Natural history of persistent HBV infection after neonatal or adult exposure. During conversion from HBe antigene-mia to the presence of anti-HBe, viremia ceases. The hepatitis associated with this seroconversion is the result of immune lysis of hepatocytes supporting HBV replication. This immune pressure may select an HBe-negative strain of HBV which causes viremia in the absence of HBeAg. AST, Aspartate aminotransferase (an indication of lysis of hepatocytes).

Years

Figure 3 Natural history of persistent HBV infection after neonatal or adult exposure. During conversion from HBe antigene-mia to the presence of anti-HBe, viremia ceases. The hepatitis associated with this seroconversion is the result of immune lysis of hepatocytes supporting HBV replication. This immune pressure may select an HBe-negative strain of HBV which causes viremia in the absence of HBeAg. AST, Aspartate aminotransferase (an indication of lysis of hepatocytes).

Seroconversion Latent HBeAg-negalive hepatitis I Infection jviremla

HBV (DNA, DNA polymerase)

HBV (DNA, DNA polymerase)

Seroconversion Latent HBeAg-negalive hepatitis I Infection jviremla

HBeAg/Ab

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