How to get rid of striae naturally
Stretch Marks Prevention and Treatment. Learn What Exactly Are Stretch Marks And How Can They Be Treated. MP3 Audio included for your PC or IPod.
Just as for assessing treatment benefits, aggregate data on side-effects can be misleading. Thus, when comparing two types of corticosteroids, one may find that the mean decrease in skin thickness is similar in the two groups. Yet, if by further scrutiny of the individual data, one finds that two children in one group developed skin thinning with noticeable visible striae, then that might influence your decision to use such treatment despite the relative reassurance of the group means. Even when such adverse events are very rare, this may be of little comfort to the patient in the sense that it is an all-or-nothing predetermined event.
Within the auditory nerve are two types of fibre, vestibular and cochlear hence its alternative name. The nerve is formed in the internal meatus and then passes medially to join the brain stem at the cerebromedullary angle. Cochlear fibres arising from the bipolar spiral ganglion cells of the cochlea pass to the dorsal and ventral cochlear nuclei in the upper medulla. Efferent fibres cross to the opposite side to form the auditory striae in the floor of the fourth ventricle. Those from the ventral nucleus particularly forming the trapezoid body in the pons. Fibres ascend from the trapezoid body in the lateral lemniscus to reach the medial geniculate body and thence to the auditory cortex. Vestibular fibres arising from the semicircular ducts, saccule and utricle pass to the vestinbular ganglion in the internal meatus and ultimately terminate in the vestibular nuclei in the floor of the fourth ventricle. Efferent fibres travel to the cerebellum in the inferior cerebellar peduncle and...
LICHEN PLANUS Lichen planus is one of a group of chronic cutaneous vesiculoerosive diseases affecting approximately 1 percent of the population. Females are affected three times as often as males. Certain drugs such as ACE inhibitors have been shown to cause lichenoid-type reactions, and these must be differentiated from lichen planus. Lichen planus is a result of T-lymphocyte attack on the basal cell layer. Approximately, 50 percent of patients with cutaneous lesions have oral lesions, but only 25 percent of patients have isolated oral lesions. Lichen planus can affect any mucosal surface however, orally, the buccal mucosa is most common (Fig 234-14). In its reticular form, lichen planus appears as multiple scattered white papules interconnected via white lines called Wickham's striae. Definitive diagnosis is via biopsy and immunofluorescent staining. The reticular and papular patterns are usually asymptomatic and require no treatment. Symptomatic erosive and atrophic forms of lichen...
Measurements of height and weight, signs of other illnesses, and evidence of cachexia should be assessed. The skin, breasts, and genital tissues should be evaluated for estrogen deficiency. The breasts should be palpated, including an attempt to express galactorrhea. The skin should be examined for hirsutism, acne, striae, acanthosis nigricans, vitiligo, thickness or thinness, and easy bruisability.
Clinically the patient presents with truncal obesity, moon face, hypertension, diabetes, abdominal striae, acne and a buffalo hump, and experiences profound weakness. Pituitary ACTH-producing tumours tend also to produce skin pigmentation as ACTH has a similar molecular structure to melanocyte-stimulating hormone (MSH). Because patients have a tendency to bruise easily with delicate skin which is easily damaged, and have an increased risk of infection, post-operative problems are increased.
Physical findings include truncal obesity with peripheral muscle wasting as in Cushing's syndrome, virilization in women or feminization in men, abdominal striae, and left varicocele. Virilization in women can include hirsutism, clitoromegaly, severe thinning of the hair, and deepening of the voice. Feminization in men is usually manifested by breast enlargement, softening of the skin, and frequent mood changes. The above physical findings are all secondary to excess sex hormone production. It is unusual to palpate an adrenocortical cancer since truncal obesity can often occur. However, a patient may present with a left varicocele from occlusion of the left spermatic vein, or even bilateral lower extremity edema secondary to obstruction of the inferior vena cava.
Dissection involving the ascending aorta is an absolute indication for operation to replace the aortic root in Marfan syndrome. It is said that at surgery there may be evidence of previous healed dissections, suggesting previous episodes of dissection have been survived. Indeed, there may be scars and stretch marks on the intima of the very attenuated sinuses of a Marfan aorta, but I do not believe that aortic dissection of the type we diagnose characteristically in these patients (figs 21.2 and 21.3) heals back to a subtle intimal lesion.
During the early years of motoring, forensic experts had already addressed the injuries characteristic of the recumbent body being run over. However, regular tire imprints on the victim's body are rarely observed and occur only at low speeds (e.g., when the victim is run over by a vehicle that is backing up). Contrary to popular belief, the most common traces found only resemble tire marks or are completely noncharacteristic (Fig. 2). The parallel, concentrated striae-like skin ruptures resulting from excessive pressure may also indicate the rolling of the victim's body by the wheel (Fig. 3). However, these signs are not specific for runover cases and are also likely to be caused by hitting the pedestrian in the standing position, because the rapid movement of the lower extremities may lead to similar skin tensions and ruptures, especially in groin region (3,8,9). Fig. 3. Striae-like skin ruptures. Fig. 3. Striae-like skin ruptures.
Cushing defined a syndrome he called pituitary basophilism. Muscular weakness, truncal obesity, abdominal striae, diabetes and hypertension characterized this clinical complex. Today, the condition resulting from chronic glucocorticoid excess due to any source is known as Cushing's syndrome, as opposed to the pituitary-derived Cushing's disease. A high index of suspicion is necessary to make the diagnosis of Cushing's syndrome early in its course. A constellation of symptoms and signs are recognized. These include a characteristic round facies, proximal muscle weakness, truncal obesity, buffalo hump, thin skin, hirsutism, easy bruisability, purple striae, depression, osteoporosis, hypertension, and glucose intolerance (Table 18.1). Purple abdominal striae
Skin problems Intertrigo, seborrheic eczema, and thrush are common in the thick heavy skinfolds of severely obese children. Pink or pale cutaneous striae, distinct from the purplish striae resulting from thinning of subcutaneous tissues in Cushing's syndrome, are common on the abdomen and upper limbs and may be a source of embarrassment. Hirsutes (abnormal facial and body hair) occurs particularly in adolescent girls with polycystic ovarian syndrome, which is associated with obesity and insulin resistance. Acanthosis nigricans, a velvety, pigmented, thickening of the skin usually at the back of the neck, is another important marker for insulin resistance, affecting up to 90 of children with type 2 diabetes mellitus.
Cushing syndrome results from exogenous steroid administration or excess endogenous cortisol secretion. The clinical manifestations of Cushing syndrome include HTN, edema, muscle weakness, glucose intolerance, osteoporosis, easy bruising, cutaneous striae, and truncal obesity (buffalo hump, moon facies). Women may develop acne, hirsutism, and amenorrhea as a result of adrenal androgen excess.
Autoimmune hepatitis (AH) is now the preferred name for the disease originally described as chronic active hepatitis (CAH) in the 1940s in reference to a protracted viral hepatitis among military personnel. However, CAH, as later understood, emerged from descriptions in the 1950s of a relapsing or progressive and usually fatal liver disease of young women of no ascertainable cause, but perhaps the result of subclinical viral hepatitis. Emphasis was given to endocrine abnormality, amenorrhea, striae and acne due to the diseased liver failing to metabolize steroid hormones, and to hyperglobulinemia with reversal of the albumin-globulin ratio in serum, a then popular liver functional index. Autoimmunity was later implicated because of findings of lupus erythematosus (LE) cells in the blood, and a positive serum complement fixation test with human tissue homogenates as antigen. Clinical and serological resemblances between CAH and systemic lupus erythematosus (SLE) led to the name lupoid...