results in a complex series of events resulting in the release of renin from the kidneys and the subsequent formation of angiotensin II and the minerocorticoid, aldosterone. Angiotensin II is a potent vasoconstrictor and stimulator of thirst. Aldosterone promotes sodium resorption, which allows the blood to retain more water. The net result of these regulatory mechanisms is concentrated urine and maintenance of plasma volume, provided that exogenous fluid intake increases proportionally. If fluid intake is not increased, dehydration will still result.

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